SLC2A2 (P11168) variants and mutations

SLC2A2 (also known as P11168) is a human protein-coding gene encoding a solute carrier family 2, facilitated glucose transporter member 2 protein. It enables high-capacity bidirectional glucose transport in liver, intestine, kidney, and pancreatic cells, matching transport to changing glucose concentrations. Biallelic loss-of-function variants cause Fanconi-Bickel syndrome with hepatomegaly, abnormal glucose homeostasis, and renal tubular dysfunction. This analysis covers 836 SLC2A2 variants and mutations. Of these, 76% have computational variant effect predictions. Disease context includes glycogen storage disease due to GLUT2 deficiency, type 2 diabetes mellitus, and diabetic ketoacidosis. Example SLC2A2 variants include M1R, T2P, and T2R.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable SLC2A2 variants

Examples include M1R, T2P, T2R, E3*, E3A, D4H, K5*, K5E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.