SLC2A1 (P11166) variants and mutations

SLC2A1 (also known as P11166) is a human protein-coding gene encoding a solute carrier family 2, facilitated glucose transporter member 1 protein. It provides basal glucose uptake in many tissues and is the principal route for glucose entry across the blood-brain barrier. Haploinsufficiency causes GLUT1 deficiency syndrome with epilepsy, developmental impairment, and movement disorders from inadequate brain glucose delivery. This analysis covers 865 SLC2A1 variants and mutations. Of these, 77% have computational variant effect predictions. Disease context includes encephalopathy due to GLUT1 deficiency, Paroxysmal exertion-induced dyskinesia, and dystonia 9. Example SLC2A1 variants include M1L, M1R, and M1T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable SLC2A1 variants

Examples include M1L, M1R, M1T, M1V, E2D, P3S, S4G, S4N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.