RPE65 (Retinoid isomerohydrolase) variants and mutations

RPE65 (also known as Retinoid isomerohydrolase) is a human protein-coding gene encoding a retinoid isomerohydrolase protein. It regenerates 11-cis-retinoid chromophore in the retinal pigment epithelium, allowing visual pigments to recover after light exposure. Biallelic loss-of-function variants cause severe inherited retinal dystrophy, and RPE65-associated disease is treatable with approved gene-replacement therapy. This analysis covers 1,062 RPE65 variants and mutations. Of these, 82% have computational variant effect predictions. Disease context includes retinitis pigmentosa, Leber congenital amaurosis 2, and Leber congenital amaurosis. Example RPE65 variants include M1?, M1I, and M1T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RPE65 variants

Examples include M1?, M1I, M1T, S2Y, I3N, I3T, I3V, Q4*. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.