MYBPC3 (Myosin-binding protein C, cardiac-type) variants and mutations

The associated protein is Myosin-binding protein C, cardiac-type. A cardiac thick-filament protein positioned in the cross-bridge region of striated muscle. It binds myosin and actin and helps tune contraction, while MYBPC3 variants are a leading genetic cause of hypertrophic cardiomyopathy. This CATVariant analysis covers 2,593 MYBPC3 variants and mutations. Disease context includes hypertrophic cardiomyopathy; this analysis is associated with hypertrophic cardiomyopathy. Available evidence includes missense variants, protein structure.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, PharmGKB, MaveDB, LitVar.

Notable MYBPC3 variants

Examples include M1?, M1I, M1L, P2R, P2T, P4L, P4S, G5R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.