LBR (Delta(14)-sterol reductase LBR) variants and mutations

LBR (also known as Delta(14)-sterol reductase LBR) is a human protein-coding gene encoding a delta(14)-sterol reductase protein. An inner nuclear-membrane protein with sterol-reductase activity in the cholesterol-biosynthesis pathway. It also contributes to nuclear-envelope organization and myeloid-cell maturation, and LBR variants are associated with Pelger-Huet anomaly and skeletal dysplasia. This analysis covers 1,020 LBR variants and mutations. Of these, 98% have computational variant effect predictions. Disease context includes Greenberg dysplasia, regressive spondylometaphyseal dysplasia, and Pelger-Huet anomaly. Example LBR variants include M1V, P2L, and P2Q.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, PharmGKB, MaveDB, LitVar.

Notable LBR variants

Examples include M1V, P2L, P2Q, S3G, S3N, S3T, R4G, R4M. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.