KLF4 (Krueppel-like factor 4) variants and mutations

KLF4 (also known as Krueppel-like factor 4) is a human protein-coding gene encoding a krueppel-like factor 4 protein. It regulates transcriptional programs controlling epithelial differentiation, stem-cell state, cell-cycle arrest, and tissue homeostasis. Depending on cellular context it can act as a tumor suppressor or oncogenic factor, and it is one of the classic factors capable of reprogramming somatic cells to pluripotency. This analysis covers 7 KLF4 variants and mutations. Of these, 29% have computational variant effect predictions. Disease context includes neurodegenerative disease, prostate carcinoma, and breast carcinoma. Example KLF4 variants include G85S, S143A, and G174E.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable KLF4 variants

Examples include G85S, S143A, G174E, N296K, T315S, L321F, T429A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.