GJB1 (Gap junction beta-1 protein) variants and mutations

GJB1 (also known as Gap junction beta-1 protein) is a human protein-coding gene encoding a gap junction beta-1 protein. It forms connexin 32 channels that support metabolic and signaling communication across layers of peripheral myelin produced by Schwann cells. Pathogenic variants cause X-linked Charcot-Marie-Tooth disease type 1, often with transient central nervous system manifestations in some individuals. This analysis covers 861 GJB1 variants and mutations. Of these, 43% have computational variant effect predictions. Disease context includes Charcot-Marie-Tooth disease X-linked dominant 1, X-linked Charcot-Marie-Tooth disease type 1, and Charcot-Marie-Tooth disease. Example GJB1 variants include M1I, M1K, and M1L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable GJB1 variants

Examples include M1I, M1K, M1L, M1R, M1T, M1V, N2K, N2S. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.