G6PC1 (P35575) variants and mutations

G6PC1 (also known as P35575) is a human protein-coding gene encoding a glucose-6-phosphatase catalytic subunit 1 protein. It catalyzes the final step of hepatic and renal glucose production by hydrolyzing glucose-6-phosphate to free glucose. Biallelic loss-of-function variants cause glycogen storage disease type Ia, with fasting hypoglycemia, lactic acidosis, hyperuricemia, hyperlipidemia, and hepatomegaly. This analysis covers 660 G6PC1 variants and mutations. Of these, 77% have computational variant effect predictions. Disease context includes glycogen storage disease due to glucose-6-phosphatase deficiency type IA, Glycogen storage disease due to glucose-6-phosphatase deficiency, and Glycogen storage disease due to glucose-6-phosphatase deficiency type a. Example G6PC1 variants include E2E, E3K, and E3E.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable G6PC1 variants

Examples include E2E, E3K, E3E, G4E, M5I, M5K, M5R, N6D. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.