DSP (Desmoplakin) variants and mutations

DSP (also known as Desmoplakin) is a human protein-coding gene encoding a desmoplakin protein. It anchors intermediate filaments to desmosomes, allowing mechanically stressed tissues such as myocardium and epidermis to maintain strong cell-cell adhesion. Pathogenic variants can cause arrhythmogenic or dilated cardiomyopathy and a range of cardiocutaneous disorders. This analysis covers 4,629 DSP variants and mutations. Of these, 75% have computational variant effect predictions. Disease context includes arrhythmogenic cardiomyopathy with wooly hair and keratoderma, arrhythmogenic right ventricular dysplasia 8, and Arrhythmogenic right ventricular dysplasia. Example DSP variants include M1R, M1V, and M1N.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable DSP variants

Examples include M1R, M1V, M1N, S2G, S2N, S2T, S2F, S2M. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.