BRIP1 (Fanconi anemia group J protein) variants and mutations

BRIP1 (also known as Fanconi anemia group J protein) is a human protein-coding gene encoding a fanconi anemia group J protein. It unwinds DNA structures and works with BRCA1 and the Fanconi-anemia pathway to repair damaged replication intermediates and interstrand crosslinks. Biallelic loss causes Fanconi anemia group J, while heterozygous loss-of-function variants increase ovarian-cancer risk. This analysis covers 5,854 BRIP1 variants and mutations. Of these, 53% have computational variant effect predictions. Disease context includes Fanconi anemia complementation group J, Fanconi anemia, and breast cancer. Example BRIP1 variants include M1L, M1V, and S2C.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable BRIP1 variants

Examples include M1L, M1V, S2C, S2F, S2T, S3*, S3L, S3T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.