ATP13A2 (Q9NQ11) variants and mutations

ATP13A2 (also known as Q9NQ11) is a human protein-coding gene encoding a polyamine-transporting ATPase 13A2 protein. It supports lysosomal and endolysosomal homeostasis and transports polyamines and other cationic substrates across intracellular membranes. Biallelic pathogenic variants cause Kufor-Rakeb syndrome and related early-onset parkinsonian-neurodegenerative phenotypes. This analysis covers 1,695 ATP13A2 variants and mutations. Of these, 81% have computational variant effect predictions. Disease context includes Kufor-Rakeb syndrome, autosomal recessive spastic paraplegia type 78, and parkinsonism due to ATP13A2 deficiency. Example ATP13A2 variants include S2C, S2N, and A3P.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable ATP13A2 variants

Examples include S2C, S2N, A3P, A3T, S5G, S5T, P7L, P7S. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.