Short-rib thoracic dysplasia 10 with or without polydactyly: genes and variants

Short-rib thoracic dysplasia 10 with or without polydactyly is linked to 1 analyzed protein (IFT172). 6 DNA variants are known to cause it; 616 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Short-rib thoracic dysplasia 10 with or without polydactyly

Known disease-causing variants in Short-rib thoracic dysplasia 10 with or without polydactyly

VariantPositionProtein partClinical label
IFT172 H1567Q1567Disease-causing (★★)
IFT172 T1623I1623Disease-causing (★★)
IFT172 R1544C1544Disease-causing (★★)
IFT172 C1727R1727Disease-causing (★★)
IFT172 Y1578C1578TPR 14Disease-causing (★)
IFT172 I411N411Disease-causing

Diseases related to Short-rib thoracic dysplasia 10 with or without polydactyly

Frequently asked questions

Which genes are linked to Short-rib thoracic dysplasia 10 with or without polydactyly?

In CATVariant, Short-rib thoracic dysplasia 10 with or without polydactyly is linked to 1 analyzed protein: IFT172 (Intraflagellar transport protein 172 homolog).

How many genetic variants are linked to Short-rib thoracic dysplasia 10 with or without polydactyly?

669 variants: 6 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 616 are of uncertain significance or have conflicting reports.

Which uncertain variants in Short-rib thoracic dysplasia 10 with or without polydactyly look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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