Pulmonary arterial hypertension associated with congenital heart disease: genes and variants

Pulmonary arterial hypertension associated with congenital heart disease is linked to 1 analyzed protein (BMPR2). 2 DNA variants are known to cause it; 3 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Pulmonary arterial hypertension associated with congenital heart disease

Weakly linked (only a few uncertain records): BMPR1A.

Known disease-causing variants in Pulmonary arterial hypertension associated with congenital heart disease

VariantPositionProtein partClinical label
BMPR2 T102A102ExtracellularDisease-causing
BMPR2 M186V186CytoplasmicDisease-causing

Same protein, different disease

Diseases related to Pulmonary arterial hypertension associated with congenital heart disease

Frequently asked questions

Which genes are linked to Pulmonary arterial hypertension associated with congenital heart disease?

In CATVariant, Pulmonary arterial hypertension associated with congenital heart disease is linked to 1 analyzed protein: BMPR2 (Bone morphogenetic protein receptor type-2).

How many genetic variants are linked to Pulmonary arterial hypertension associated with congenital heart disease?

6 variants: 2 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 3 are of uncertain significance or have conflicting reports.

Which uncertain variants in Pulmonary arterial hypertension associated with congenital heart disease look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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