Palmoplantar keratoderma, nonepidermolytic, focal 1: genes and variants

Palmoplantar keratoderma, nonepidermolytic, focal 1 is linked to 1 analyzed protein (KRT16). 6 DNA variants are known to cause it; 4 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Palmoplantar keratoderma, nonepidermolytic, focal 1

Where Palmoplantar keratoderma, nonepidermolytic, focal 1 variants cluster

Known disease-causing variants in Palmoplantar keratoderma, nonepidermolytic, focal 1

VariantPositionProtein partClinical label
KRT16 M121T121IF rodDisease-causing (★★★★)
KRT16 N125S125IF rodDisease-causing (★★)
KRT16 R127C127IF rodDisease-causing (★★)
KRT16 R127G127IF rodDisease-causing (★★)
KRT16 L124H124IF rodDisease-causing (★★)
KRT16 N125K125IF rodDisease-causing (★)

Same protein, different disease

Diseases related to Palmoplantar keratoderma, nonepidermolytic, focal 1

Frequently asked questions

Which genes are linked to Palmoplantar keratoderma, nonepidermolytic, focal 1?

In CATVariant, Palmoplantar keratoderma, nonepidermolytic, focal 1 is linked to 1 analyzed protein: KRT16 (Keratin, type I cytoskeletal 16).

How many genetic variants are linked to Palmoplantar keratoderma, nonepidermolytic, focal 1?

12 variants: 6 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 4 are of uncertain significance or have conflicting reports.

Which uncertain variants in Palmoplantar keratoderma, nonepidermolytic, focal 1 look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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