Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections: genes and variants

Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections is linked to 2 analyzed proteins (FBN1 and LOX). 10 DNA variants are known to cause it; 1 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections

Known disease-causing variants in Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections

VariantPositionProtein partClinical label
FBN1 C166Y166EGF-like 3Disease-causing (★★)
FBN1 C750Y750EGF-like 11Disease-causing (★★)
FBN1 C1672R1672EGF-like 28Disease-causing (★★)
FBN1 C1905S1905EGF-like 32Disease-causing (★★)
FBN1 C358S358TB 2Disease-causing (★★)
FBN1 C1695W1695TB 7Disease-causing (★★)
FBN1 C1818R1818EGF-like 30Disease-causing (★★)
FBN1 C2522Y2522EGF-like 43Disease-causing (★★)
FBN1 C862S862TB 4Disease-causing (★)
FBN1 C1674S1674EGF-like 28Disease-causing (★)

Same protein, different disease

Diseases related to Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections

Frequently asked questions

Which genes are linked to Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections?

In CATVariant, Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections is linked to 2 analyzed proteins: FBN1 (Fibrillin-1) and LOX (Protein-lysine 6-oxidase).

How many genetic variants are linked to Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections?

17 variants: 10 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 1 are of uncertain significance or have conflicting reports.

Which uncertain variants in Marfan syndrome/loeys-dietz syndrome/familial thoracic aortic aneurysms and dissections look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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