Hyperlipidemia, familial combined, LPL related: genes and variants

Hyperlipidemia, familial combined, LPL related is linked to 1 analyzed protein (LPL). 18 DNA variants are known to cause it; 21 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Hyperlipidemia, familial combined, LPL related

Known disease-causing variants in Hyperlipidemia, familial combined, LPL related

VariantPositionProtein partClinical label
LPL R270C270Disease-causing (★★)
LPL M1I1Disease-causing (★★)
LPL W113R113Disease-causing (★★)
LPL H268Y268Disease-causing (★★)
LPL M328I328Disease-causing (★★)
LPL A98T98Disease-causing (★★)
LPL D183N183Disease-causing (★★)
LPL G215E215Disease-causing (★★)
LPL P234L234Disease-causing (★★)
LPL I252T252Essential for determining substrate specificityDisease-causing (★★)
LPL D277N277Disease-causing (★★)
LPL L279V279Disease-causing (★★)
LPL C302R302Disease-causing (★)
LPL D231N231Disease-causing (★)
LPL C310Y310Disease-causing (★)
LPL A361T361PLATDisease-causing (★)
LPL G436R436PLATDisease-causing (★)
LPL R197H197Disease-causing

Same protein, different disease

Diseases related to Hyperlipidemia, familial combined, LPL related

Frequently asked questions

Which genes are linked to Hyperlipidemia, familial combined, LPL related?

In CATVariant, Hyperlipidemia, familial combined, LPL related is linked to 1 analyzed protein: LPL (Lipoprotein lipase).

How many genetic variants are linked to Hyperlipidemia, familial combined, LPL related?

40 variants: 18 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 21 are of uncertain significance or have conflicting reports.

Which uncertain variants in Hyperlipidemia, familial combined, LPL related look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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