Fanconi-Bickel syndrome: genes and variants

Fanconi-Bickel syndrome is linked to 1 analyzed protein (SLC2A2). 10 DNA variants are known to cause it; 82 more are uncertain, and 1 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Fanconi-Bickel syndrome

Known disease-causing variants in Fanconi-Bickel syndrome

VariantPositionProtein partClinical label
SLC2A2 V197L197TransmembraneDisease-causing (★★)
SLC2A2 P417L417TransmembraneDisease-causing (★★)
SLC2A2 R158S158ExtracellularDisease-causing (★★)
SLC2A2 G318R318TransmembraneDisease-causing (★)
SLC2A2 W444R444TransmembraneDisease-causing (★)
SLC2A2 G416S416TransmembraneDisease-causing (★)
SLC2A2 M1R1CytoplasmicDisease-causing (★)
SLC2A2 G20D20TransmembraneDisease-causing (★)
SLC2A2 V423E423CytoplasmicDisease-causing
SLC2A2 L389P389TransmembraneDisease-causing

Uncertain variants in Fanconi-Bickel syndrome that look disease-causing

VariantPositionProtein partClinical labelEvidence
SLC2A2 P417Q417TransmembraneUncertain (★)+6: 2 other pathogenic changes within 3 positions; P417L at the same position is pathogenic; not seen in the gnomAD population database; AlphaMissense 0.70

Diseases related to Fanconi-Bickel syndrome

Frequently asked questions

Which genes are linked to Fanconi-Bickel syndrome?

In CATVariant, Fanconi-Bickel syndrome is linked to 1 analyzed protein: SLC2A2 (Solute carrier family 2, facilitated glucose transporter member 2).

How many genetic variants are linked to Fanconi-Bickel syndrome?

96 variants: 10 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 82 are of uncertain significance or have conflicting reports.

Which uncertain variants in Fanconi-Bickel syndrome look disease-causing?

1 uncertain variants reach the likely-pathogenic range of the ACMG/AMP points scale on computable evidence, for example SLC2A2 P417Q. These are leads for expert review, not diagnoses.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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