Factor VII deficiency: genes and variants

Factor VII deficiency is linked to 1 analyzed protein (F7). 8 DNA variants are known to cause it; 9 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: congenital factor VII deficiency

Genes linked to Factor VII deficiency

Weakly linked (only a few uncertain records): F8.

Where Factor VII deficiency variants cluster

Known disease-causing variants in Factor VII deficiency

VariantPositionProtein partClinical label
F7 R364W364Peptidase S1Disease-causing (★★)
F7 R337C337Peptidase S1Disease-causing (★★)
F7 R364Q364Peptidase S1Disease-causing (★★)
F7 A429T429Peptidase S1Disease-causing (★★)
F7 A354V354Peptidase S1Disease-causing (★★)
F7 A304V304Peptidase S1Disease-causing (★★)
F7 R307H307Peptidase S1Disease-causing (★★)
F7 N117D117EGF-like 1Disease-causing

Which prediction tools work for Factor VII deficiency

How often each tool ranks a disease-causing variant above a harmless one (AUROC × 100).

Diseases related to Factor VII deficiency

Frequently asked questions

Which genes are linked to Factor VII deficiency?

In CATVariant, Factor VII deficiency is linked to 1 analyzed protein: F7 (Coagulation factor VII).

How many genetic variants are linked to Factor VII deficiency?

93 variants: 8 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 9 are of uncertain significance or have conflicting reports.

Which uncertain variants in Factor VII deficiency look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

Which variant effect predictor works best for Factor VII deficiency?

Among tools not trained on clinical labels, SIFT separates this disease's known disease-causing variants from harmless ones best (AUROC 0.84, based on 8 disease-causing and 40 harmless variants).

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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