Diabetes insipidus, nephrogenic, autosomal: genes and variants

Diabetes insipidus, nephrogenic, autosomal is linked to 1 analyzed protein (AQP2). 16 DNA variants are known to cause it; 38 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Diabetes insipidus, nephrogenic, autosomal

Where Diabetes insipidus, nephrogenic, autosomal variants cluster

Known disease-causing variants in Diabetes insipidus, nephrogenic, autosomal

VariantPositionProtein partClinical label
AQP2 G64R64CytoplasmicDisease-causing (★★)
AQP2 G100V100TransmembraneDisease-causing (★★)
AQP2 V71M71Discontinuously helicalDisease-causing (★★)
AQP2 R187H187Discontinuously helicalDisease-causing (★★)
AQP2 Q57P57TransmembraneDisease-causing (★★)
AQP2 A147T147TransmembraneDisease-causing (★★)
AQP2 V168M168TransmembraneDisease-causing (★★)
AQP2 P185A185Discontinuously helicalDisease-causing (★★)
AQP2 T126M126ExtracellularDisease-causing (★★)
AQP2 T125M125ExtracellularDisease-causing (★★)
AQP2 P262L262CytoplasmicDisease-causing (★★)
AQP2 A130V130TransmembraneDisease-causing (★)
AQP2 G100R100TransmembraneDisease-causing
AQP2 N68S68Discontinuously helicalDisease-causing
AQP2 S188F188Discontinuously helicalDisease-causing
AQP2 C181W181Discontinuously helicalDisease-causing

Same protein, different disease

Diseases related to Diabetes insipidus, nephrogenic, autosomal

Frequently asked questions

Which genes are linked to Diabetes insipidus, nephrogenic, autosomal?

In CATVariant, Diabetes insipidus, nephrogenic, autosomal is linked to 1 analyzed protein: AQP2 (Aquaporin-2).

How many genetic variants are linked to Diabetes insipidus, nephrogenic, autosomal?

75 variants: 16 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 38 are of uncertain significance or have conflicting reports.

Which uncertain variants in Diabetes insipidus, nephrogenic, autosomal look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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