Autosomal recessive severe congenital neutropenia due to CSF3R deficiency: genes and variants

Autosomal recessive severe congenital neutropenia due to CSF3R deficiency is linked to 1 analyzed protein (CSF3R). 2 DNA variants are known to cause it; 303 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to Autosomal recessive severe congenital neutropenia due to CSF3R deficiency

Known disease-causing variants in Autosomal recessive severe congenital neutropenia due to CSF3R deficiency

VariantPositionProtein partClinical label
CSF3R R308C308Fibronectin type-III 2Disease-causing (★★)
CSF3R T618I618Fibronectin type-III 5Disease-causing (★★)

Diseases related to Autosomal recessive severe congenital neutropenia due to CSF3R deficiency

Frequently asked questions

Which genes are linked to Autosomal recessive severe congenital neutropenia due to CSF3R deficiency?

In CATVariant, Autosomal recessive severe congenital neutropenia due to CSF3R deficiency is linked to 1 analyzed protein: CSF3R (Granulocyte colony-stimulating factor receptor).

How many genetic variants are linked to Autosomal recessive severe congenital neutropenia due to CSF3R deficiency?

330 variants: 2 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 303 are of uncertain significance or have conflicting reports.

Which uncertain variants in Autosomal recessive severe congenital neutropenia due to CSF3R deficiency look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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