ATP1A3-associated neurological disorder: genes and variants

ATP1A3-associated neurological disorder is linked to 1 analyzed protein (ATP1A3). 4 DNA variants are known to cause it; 0 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to ATP1A3-associated neurological disorder

Known disease-causing variants in ATP1A3-associated neurological disorder

VariantPositionProtein partClinical label
ATP1A3 R756H756CytoplasmicDisease-causing (★★)
ATP1A3 I777N777TransmembraneDisease-causing (★★)
ATP1A3 D923N923TransmembraneDisease-causing (★★)
ATP1A3 S359Y359CytoplasmicDisease-causing (★)

Same protein, different disease

Diseases related to ATP1A3-associated neurological disorder

Frequently asked questions

Which genes are linked to ATP1A3-associated neurological disorder?

In CATVariant, ATP1A3-associated neurological disorder is linked to 1 analyzed protein: ATP1A3 (Sodium/potassium-transporting ATPase subunit alpha-3).

How many genetic variants are linked to ATP1A3-associated neurological disorder?

7 variants: 4 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 0 are of uncertain significance or have conflicting reports.

Which uncertain variants in ATP1A3-associated neurological disorder look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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