ATP1A3 (P13637) variants and mutations

ATP1A3 (also known as P13637) is a human protein-coding gene encoding a sodium/potassium-transporting ATPase subunit alpha-3 protein. It rapidly restores neuronal sodium and potassium gradients after repetitive firing, making it particularly important in highly active neurons. Pathogenic variants cause overlapping syndromes including alternating hemiplegia of childhood, rapid-onset dystonia-parkinsonism, and CAPOS syndrome. This analysis covers 827 ATP1A3 variants and mutations. Of these, 74% have computational variant effect predictions. Disease context includes alternating hemiplegia of childhood 2, dystonia 12, and Cerebellar ataxia - areflexia - pes cavus - optic atrophy - sensorineural hearin. Example ATP1A3 variants include G2A, G2R, and G2V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable ATP1A3 variants

Examples include G2A, G2R, G2V, G2W, D3G, K5E, D6E, D7E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.