3-Methylglutaconic aciduria type 2: genes and variants

3-Methylglutaconic aciduria type 2 is linked to 1 analyzed protein (TAFAZZIN). 12 DNA variants are known to cause it; 105 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to 3-Methylglutaconic aciduria type 2

Known disease-causing variants in 3-Methylglutaconic aciduria type 2

VariantPositionProtein partClinical label
TAFAZZIN R94C94Mitochondrial intermembraneDisease-causing (★★)
TAFAZZIN R94G94Mitochondrial intermembraneDisease-causing (★★)
TAFAZZIN R94S94Mitochondrial intermembraneDisease-causing (★★)
TAFAZZIN R94L94Mitochondrial intermembraneDisease-causing (★★)
TAFAZZIN R94H94Mitochondrial intermembraneDisease-causing (★★)
TAFAZZIN G186R186Mitochondrial targeting sequenceDisease-causing (★★)
TAFAZZIN G80R80Mitochondrial intermembraneDisease-causing (★★)
TAFAZZIN C103R103Mitochondrial intermembraneDisease-causing (★)
TAFAZZIN G124R124Mitochondrial intermembraneDisease-causing (★)
TAFAZZIN C72F72HXXXXD motifDisease-causing (★)
TAFAZZIN D74E74HXXXXD motifDisease-causing (★)
TAFAZZIN F148I148Mitochondrial intermembraneDisease-causing (★)

Diseases related to 3-Methylglutaconic aciduria type 2

Frequently asked questions

Which genes are linked to 3-Methylglutaconic aciduria type 2?

In CATVariant, 3-Methylglutaconic aciduria type 2 is linked to 1 analyzed protein: TAFAZZIN (Tafazzin).

How many genetic variants are linked to 3-Methylglutaconic aciduria type 2?

117 variants: 12 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 105 are of uncertain significance or have conflicting reports.

Which uncertain variants in 3-Methylglutaconic aciduria type 2 look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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