TSC1 (Hamartin) variants and mutations

TSC1 (also known as Hamartin) is a human protein-coding gene encoding a hamartin protein. Together with TSC2, it restrains RHEB and mTORC1 signaling, preventing inappropriate cell growth when nutrients or growth signals are limited. Loss-of-function variants cause tuberous sclerosis complex with hamartomas and tumors in the brain, kidney, skin, heart, lungs, and other organs. This analysis covers 5,123 TSC1 variants and mutations. Of these, 47% have computational variant effect predictions. Disease context includes tuberous sclerosis, isolated focal cortical dysplasia type II, and tuberous sclerosis 1. Example TSC1 variants include M1?, M1I, and A2T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable TSC1 variants

Examples include M1?, M1I, A2T, A2V, Q3*, Q3R, Q4*, Q4H. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.