TNNT2 (Troponin T, cardiac muscle) variants and mutations

TNNT2 (also known as Troponin T, cardiac muscle) is a human protein-coding gene encoding a troponin T, cardiac muscle protein. It anchors the cardiac troponin complex to tropomyosin and helps translate calcium-dependent conformational changes into controlled actin-myosin interaction. Pathogenic variants can cause hypertrophic, dilated, or restrictive cardiomyopathy and alter arrhythmic risk. This analysis covers 566 TNNT2 variants and mutations. Of these, 90% have computational variant effect predictions. Disease context includes hypertrophic cardiomyopathy, left ventricular noncompaction, and hypertrophic cardiomyopathy 2. Example TNNT2 variants include M1I, M1V, and S2F.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable TNNT2 variants

Examples include M1I, M1V, S2F, S2P, D3N, I4L, I4M, I4V. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.