TNNI3 (Troponin I, cardiac muscle) variants and mutations

TNNI3 (also known as Troponin I, cardiac muscle) is a human protein-coding gene encoding a troponin I, cardiac muscle protein. It restrains cardiac actin-myosin interaction at low calcium and shifts position within the troponin complex when calcium binds, allowing contraction. Pathogenic variants can alter thin-filament calcium sensitivity and cause hypertrophic, restrictive, or dilated cardiomyopathy. This analysis covers 672 TNNI3 variants and mutations. Of these, 92% have computational variant effect predictions. Disease context includes hypertrophic cardiomyopathy, cardiomyopathy, familial restrictive, 1, and dilated cardiomyopathy 1FF. Example TNNI3 variants include M1I, M1V, and A2E.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable TNNI3 variants

Examples include M1I, M1V, A2E, A2S, A2T, A2V, D3E, D3N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.