SMARCA4 (P51532) variants and mutations

SMARCA4 (also known as P51532) is a human protein-coding gene encoding a SWI/SNF-related matrix-associated actin-dependent regulator of chromatin subfamily A member 4 protein. Its ATPase activity drives nucleosome remodeling in BAF-family complexes and thereby controls access to regulatory DNA. Germline pathogenic variants cause Coffin-Siris syndrome or rhabdoid-tumor predisposition, while somatic loss defines several aggressive cancers. This analysis covers 7,890 SMARCA4 variants and mutations. Of these, 37% have computational variant effect predictions. Disease context includes intellectual disability, autosomal dominant 16, rhabdoid tumor predisposition syndrome 2, and familial rhabdoid tumor. Example SMARCA4 variants include M1I, S2C, and S2F.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable SMARCA4 variants

Examples include M1I, S2C, S2F, S2P, S2T, S2Y, T3A, T3I. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.