SETX (Helicase senataxin) variants and mutations

SETX (also known as Helicase senataxin) is a human protein-coding gene encoding a helicase senataxin protein. It resolves RNA-DNA hybrids and supports transcription termination, RNA processing, and genome stability, particularly in long-lived neurons. Different pathogenic mechanisms cause ataxia with oculomotor apraxia type 2 or juvenile amyotrophic lateral sclerosis type 4. This analysis covers 3,603 SETX variants and mutations. Of these, 76% have computational variant effect predictions. Disease context includes spinocerebellar ataxia, autosomal recessive, with axonal neuropathy 2, amyotrophic lateral sclerosis type 4, and Spinocerebellar ataxia with axonal neuropathy type 2. Example SETX variants include S2G, S2R, and T3A.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable SETX variants

Examples include S2G, S2R, T3A, T3I, C4R, C5S, W6L, C7Y. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.