SETD2 (Q9BYW2) variants and mutations

SETD2 (also known as Q9BYW2) is a human protein-coding gene encoding a histone-lysine N-methyltransferase protein. It deposits H3K36 trimethylation across actively transcribed genes and helps coordinate RNA processing, DNA repair, and genome stability. Somatic loss is common in renal and other cancers, while germline pathogenic variants can cause Luscan-Lumish overgrowth syndrome. This analysis covers 10,492 SETD2 variants and mutations. Of these, 26% have computational variant effect predictions. Disease context includes Luscan-Lumish syndrome, clear cell renal carcinoma, and pleural mesothelioma. Example SETD2 variants include M1K, M1T, and K2N.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable SETD2 variants

Examples include M1K, M1T, K2N, K2R, Q3*, Q3E, L4V, Q5*. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.