SDHA (P31040) variants and mutations

SDHA (also known as P31040) is a human protein-coding gene encoding a succinate dehydrogenase [ubiquinone] flavoprotein subunit, mitochondrial protein. It catalyzes oxidation of succinate to fumarate while transferring electrons into respiratory-chain complex II, directly linking the TCA cycle with oxidative phosphorylation. Biallelic deficiency can cause mitochondrial disease, while heterozygous loss-of-function variants predispose to paraganglioma, pheochromocytoma, and selected gastrointestinal stromal tumors. This analysis covers 1,844 SDHA variants and mutations. Of these, 76% have computational variant effect predictions. Disease context includes mitochondrial complex II deficiency, nuclear type 1, pheochromocytoma/paraganglioma syndrome 5, and dilated cardiomyopathy 1GG. Example SDHA variants include M1?, M1I, and M1K.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable SDHA variants

Examples include M1?, M1I, M1K, M1L, M1R, M1T, S2*, S2A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.