RUNX1 (Q01196) variants and mutations

RUNX1 (also known as Q01196) is a human protein-coding gene encoding a runt-related transcription factor 1 protein. It controls hematopoietic stem-cell emergence and later megakaryocytic and myeloid differentiation through lineage-specific transcriptional programs. Germline loss-of-function variants cause familial platelet disorder with myeloid malignancy predisposition, while somatic mutations and fusions are common in leukemia. This analysis covers 1,893 RUNX1 variants and mutations. Of these, 45% have computational variant effect predictions. Disease context includes hereditary thrombocytopenia and hematological cancer predisposition syndrome ass, acute myeloid leukemia, and hereditary thrombocytopenia and hematologic cancer predisposition syndrome. Example RUNX1 variants include M1?, R2C, and R2G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RUNX1 variants

Examples include M1?, R2C, R2G, R2H, I3S, V5I, V5L, D6A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.