RECQL4 (ATP-dependent DNA helicase Q4) variants and mutations

RECQL4 (also known as ATP-dependent DNA helicase Q4) is a human protein-coding gene encoding an ATP-dependent DNA helicase Q4 protein. It participates in DNA replication, repair, and maintenance of genome stability, particularly during replication initiation and processing of damaged DNA. Biallelic pathogenic variants cause Rothmund-Thomson, Baller-Gerold, or RAPADILINO syndromes, with variable skeletal abnormalities and cancer predisposition. This analysis covers 3,711 RECQL4 variants and mutations. Of these, 76% have computational variant effect predictions. Disease context includes Rothmund-Thomson syndrome type 2, Baller-Gerold syndrome, and rapadilino syndrome. Example RECQL4 variants include M1?, M1I, and M1L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RECQL4 variants

Examples include M1?, M1I, M1L, M1T, M1V, E2G, E2Q, E2V. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.