RAD50 (DNA repair protein RAD50) variants and mutations

RAD50 (also known as DNA repair protein RAD50) is a human protein-coding gene encoding a DNA repair protein. It provides ATP-dependent DNA tethering within the MRE11-RAD50-NBN complex and helps organize detection and processing of double-strand breaks. Biallelic loss-of-function variants can cause a Nijmegen-breakage-syndrome-like disorder with chromosome instability and developmental abnormalities. This analysis covers 3,347 RAD50 variants and mutations. Of these, 73% have computational variant effect predictions. Disease context includes Nijmegen breakage syndrome-like disorder, cancer, and Inherited cancer-predisposing syndrome. Example RAD50 variants include M1I, M1V, and S2A.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RAD50 variants

Examples include M1I, M1V, S2A, S2F, S2P, S2T, S2S, R3G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.