PMS2 (P54278) variants and mutations

PMS2 (also known as P54278) is a human protein-coding gene encoding a mismatch repair endonuclease protein. Together with MLH1, it provides endonuclease activity needed to complete DNA mismatch repair after replication errors are recognized. Germline loss-of-function variants cause Lynch syndrome, while biallelic variants cause constitutional mismatch-repair deficiency. This analysis covers 4,022 PMS2 variants and mutations. Of these, 66% have computational variant effect predictions. Disease context includes Lynch syndrome, Constitutional mismatch repair deficiency syndrome, and mismatch repair cancer syndrome 1. Example PMS2 variants include M1?, M1I, and M1K.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable PMS2 variants

Examples include M1?, M1I, M1K, M1L, M1R, M1T, M1V, E2*. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.