NTHL1 (Endonuclease III-like protein 1) variants and mutations

NTHL1 (also known as Endonuclease III-like protein 1) is a human protein-coding gene encoding an endonuclease III-like protein 1 protein. It removes oxidized pyrimidines from DNA through base-excision repair and prevents accumulation of characteristic point mutations. Biallelic loss-of-function variants cause NTHL1 tumor syndrome with colorectal polyposis and increased risk of multiple malignancies. This analysis covers 1,074 NTHL1 variants and mutations. Of these, 82% have computational variant effect predictions. Disease context includes familial adenomatous polyposis 3, attenuated familial adenomatous polyposis, and cancer. Example NTHL1 variants include M1I, M1K, and M1L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable NTHL1 variants

Examples include M1I, M1K, M1L, M1T, M1V, T2A, T2I, T2N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.