MYLK (Q15746) variants and mutations

MYLK (also known as Q15746) is a human protein-coding gene encoding a myosin light chain kinase, smooth muscle protein. It phosphorylates myosin regulatory light chains to initiate smooth-muscle contraction in blood vessels and visceral tissues. Pathogenic loss-of-function variants can reduce arterial contractile integrity and cause familial thoracic aortic aneurysm and dissection. This analysis covers 2,639 MYLK variants and mutations. Of these, 71% have computational variant effect predictions. Disease context includes aortic aneurysm, familial thoracic 7, familial thoracic aortic aneurysm and aortic dissection, and megacystis-microcolon-intestinal hypoperistalsis syndrome 1. Example MYLK variants include M1V, G2E, and D3N.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable MYLK variants

Examples include M1V, G2E, D3N, D3V, D3Y, K5N, L6P, V7A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.