MYL2 (P10916) variants and mutations

MYL2 (also known as P10916) is a human protein-coding gene encoding a myosin regulatory light chain 2, ventricular/cardiac muscle isoform protein. It modulates cardiac myosin-head mechanics and phosphorylation-dependent force generation in ventricular sarcomeres. Pathogenic variants are an established cause of familial hypertrophic cardiomyopathy and can also produce other cardiomyopathy phenotypes. This analysis covers 451 MYL2 variants and mutations. Of these, 88% have computational variant effect predictions. Disease context includes hypertrophic cardiomyopathy 10, hypertrophic cardiomyopathy, and myopathy, myofibrillar, 12, infantile-onset, with cardiomyopathy. Example MYL2 variants include M1?, M1I, and M1V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable MYL2 variants

Examples include M1?, M1I, M1V, A2E, A2S, A2T, P3S, P3T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.