KCNJ5 (P48544) variants and mutations

KCNJ5 (also known as P48544) is a human protein-coding gene encoding a g protein-activated inward rectifier potassium channel 4 protein. It contributes to G-protein-activated inward-rectifier potassium current in the heart and endocrine tissues, helping regulate pacemaker activity and membrane potential. Somatic selectivity-altering variants are a common cause of aldosterone-producing adrenal adenomas, while germline variants can cause familial hyperaldosteronism. This analysis covers 992 KCNJ5 variants and mutations. Of these, 94% have computational variant effect predictions. Disease context includes familial hyperaldosteronism type III, atrial fibrillation, and aldosterone-producing adrenal cortex adenoma. Example KCNJ5 variants include A2S, A2V, and A2G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable KCNJ5 variants

Examples include A2S, A2V, A2G, G3S, G3A, G3G, D4E, D4N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.