KCNH2 (hERG) variants and mutations

KCNH2 (also known as hERG) is a human protein-coding gene encoding a voltage-gated inwardly rectifying potassium channel protein. Its unusually rapid inactivation and slow deactivation shape the rapid delayed-rectifier current IKr, a major determinant of ventricular repolarization. Loss-of-function variants can prolong repolarization and cause long-QT syndrome, whereas gain-of-function variants can cause short-QT syndrome. This analysis covers 2,412 KCNH2 variants and mutations. Of these, 90% have computational variant effect predictions. Disease context includes Romano-Ward syndrome, Familial short QT syndrome, and atrial fibrillation. Example KCNH2 variants include M1?, M1I, and P2L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable KCNH2 variants

Examples include M1?, M1I, P2L, V3L, R4Q, G6D, V8G, A9T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.