JPH2 (Junctophilin-2) variants and mutations

JPH2 (also known as Junctophilin-2) is a human protein-coding gene encoding a junctophilin-2 protein. It anchors the sarcoplasmic reticulum close to transverse tubules, creating junctional membrane domains required for tightly coupled calcium entry and calcium release in heart muscle. Pathogenic variants can disrupt excitation-contraction coupling and cause hypertrophic or dilated cardiomyopathy and inherited arrhythmia phenotypes. This analysis covers 1,526 JPH2 variants and mutations. Of these, 87% have computational variant effect predictions. Disease context includes hypertrophic cardiomyopathy 17, hypertrophic cardiomyopathy, and dilated cardiomyopathy. Example JPH2 variants include S2C, S2G, and S2N.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable JPH2 variants

Examples include S2C, S2G, S2N, S2R, S2T, G3E, G3R, R5C. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.