JAK3 (Tyrosine-protein kinase JAK3) variants and mutations

JAK3 (also known as Tyrosine-protein kinase JAK3) is a human protein-coding gene encoding a tyrosine-protein kinase protein. It carries signals from cytokine receptors using the common gamma chain and is essential for T-cell and NK-cell development. Biallelic loss-of-function variants cause severe combined immunodeficiency, while activating somatic variants occur in selected leukemias and lymphomas. This analysis covers 2,778 JAK3 variants and mutations. Of these, 53% have computational variant effect predictions. Disease context includes T-B+ severe combined immunodeficiency due to JAK3 deficiency, ulcerative colitis, and rheumatoid arthritis. Example JAK3 variants include M1?, A2P, and A2T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable JAK3 variants

Examples include M1?, A2P, A2T, A2V, P3L, P3S, S5G, S5N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.