HBA1 (Hemoglobin subunit alpha) variants and mutations

HBA1 (also known as Hemoglobin subunit alpha) is a human protein-coding gene encoding a hemoglobin subunit alpha protein. It contributes alpha-globin chains that pair with beta-like globins to carry oxygen in red blood cells. Deletion or inactivation reduces alpha-globin production and causes alpha-thalassemia, with severity determined by the number and function of affected alpha-globin genes. This analysis covers 639 HBA1 variants and mutations. Of these, 82% have computational variant effect predictions. Disease context includes hemoglobin H disease, Alpha-thalassemia, and Autosomal dominant methemoglobinemia. Example HBA1 variants include M1?, M1K, and M1R.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable HBA1 variants

Examples include M1?, M1K, M1R, M1T, M1V, V2A, V2E, V2G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.