GJA1 (Gap junction alpha-1 protein) variants and mutations

GJA1 (also known as Gap junction alpha-1 protein) is a human protein-coding gene encoding a gap junction alpha-1 protein. It forms connexin 43 gap junctions that permit direct electrical and metabolic communication between neighboring cells in heart, bone, skin, and many other tissues. Pathogenic variants cause oculodentodigital dysplasia and related syndromes with craniofacial, dental, limb, and sometimes cardiac abnormalities. This analysis covers 819 GJA1 variants and mutations. Of these, 90% have computational variant effect predictions. Disease context includes oculodentodigital dysplasia, erythrokeratodermia variabilis, and oculodentodigital dysplasia, autosomal recessive. Example GJA1 variants include M1?, G2V, and G2G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable GJA1 variants

Examples include M1?, G2V, G2G, D3G, D3Y, W4*, S5R, S5S. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.