FLCN (Folliculin) variants and mutations

FLCN (also known as Folliculin) is a human protein-coding gene encoding a folliculin protein. It couples lysosomal nutrient sensing to AMPK, mTOR, and related pathways and helps regulate cell growth and metabolism. Germline loss-of-function variants cause Birt-Hogg-Dube syndrome with fibrofolliculomas, pulmonary cysts, pneumothorax, and renal-tumor predisposition. This analysis covers 1,843 FLCN variants and mutations. Of these, 52% have computational variant effect predictions. Disease context includes Birt-Hogg-Dube syndrome 1, familial spontaneous pneumothorax, and Birt-Hogg-Dube syndrome. Example FLCN variants include M1?, M1I, and M1T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable FLCN variants

Examples include M1?, M1I, M1T, M1V, A3V, I4L, I4M, I4V. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.