FBN2 (Fibrillin-2) variants and mutations

FBN2 (also known as Fibrillin-2) is a human protein-coding gene encoding a fibrillin-2 protein. It contributes to extracellular microfibrils that guide elastic-fiber formation and tissue organization during development. Heterozygous pathogenic variants cause congenital contractural arachnodactyly, characterized by long limbs and fingers, contractures, and characteristic ear abnormalities. This analysis covers 4,797 FBN2 variants and mutations. Of these, 62% have computational variant effect predictions. Disease context includes congenital contractural arachnodactyly, Abnormality of the skeletal system, and hypertensive disorder. Example FBN2 variants include M1?, G2A, and G2E.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable FBN2 variants

Examples include M1?, G2A, G2E, G2R, G2W, R3I, R4G, R4K. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.