FBN1 (Fibrillin-1) variants and mutations

FBN1 (also known as Fibrillin-1) is a human protein-coding gene encoding a fibrillin-1 protein. Its fibrillin-1 microfibrils provide mechanical support to elastic tissues and regulate local availability of growth factors such as TGF-beta. Pathogenic variants cause Marfan syndrome and related fibrillinopathies affecting the aorta, skeleton, eyes, skin, and lungs. This analysis covers 5,677 FBN1 variants and mutations. Of these, 40% have pathogenic or likely pathogenic clinical classifications, 73% have computational variant effect predictions from REVEL and MutPred, and 37% have population-specific frequency data. Disease context includes Marfan syndrome, ectopia lentis 1, isolated, autosomal dominant, and Acromicric dysplasia. Example FBN1 variants include M1I, M1L, and M1R.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable FBN1 variants

Examples include M1I, M1L, M1R, M1T, M1V, R2C, R2G, R3*. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.