FANCC (Fanconi anemia group C protein) variants and mutations

FANCC (also known as Fanconi anemia group C protein) is a human protein-coding gene encoding a fanconi anemia group C protein. It contributes to activation of the FANCD2-FANCI DNA-repair pathway after replication-blocking lesions. Biallelic loss-of-function variants cause Fanconi anemia group C, with chromosome instability, marrow failure, congenital abnormalities, and elevated cancer risk. This analysis covers 1,516 FANCC variants and mutations. Of these, 68% have computational variant effect predictions. Disease context includes Fanconi anemia complementation group C, Fanconi anemia, and hereditary neoplastic syndrome. Example FANCC variants include M1?, M1I, and M1T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable FANCC variants

Examples include M1?, M1I, M1T, A2T, A2V, Q3E, Q3H, Q3L. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.