CRX (Cone-rod homeobox protein) variants and mutations

CRX (also known as Cone-rod homeobox protein) is a human protein-coding gene encoding a cone-rod homeobox protein. It activates photoreceptor-specific gene programs required for development and maintenance of rods and cones. Pathogenic variants can cause cone-rod dystrophy, Leber congenital amaurosis, or dominant retinitis pigmentosa depending on the molecular mechanism. This analysis covers 737 CRX variants and mutations. Of these, 80% have computational variant effect predictions. Disease context includes Leber congenital amaurosis, cone-rod dystrophy 2, and Leber congenital amaurosis 7. Example CRX variants include M2T, A3P, and A3S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable CRX variants

Examples include M2T, A3P, A3S, A3T, A3V, A3E, A3A, Y4C. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.