CREBBP (CREB-binding protein) variants and mutations

CREBBP (also known as CREB-binding protein) is a human protein-coding gene encoding a CREB-binding protein. It acetylates histones and integrates signals from many transcription factors to regulate developmental and activity-dependent gene expression. Germline loss-of-function variants cause Rubinstein-Taybi syndrome, while somatic alterations occur in several cancers. This analysis covers 8,904 CREBBP variants and mutations. Of these, 36% have computational variant effect predictions. Disease context includes Rubinstein-Taybi syndrome due to CREBBP mutations, Menke-Hennekam syndrome 1, and Rubinstein-Taybi syndrome. Example CREBBP variants include M1K, A2G, and A2S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable CREBBP variants

Examples include M1K, A2G, A2S, E3D, E3K, N4K, N4S, L6Q. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.