COL3A1 (Collagen alpha-1(III) chain) variants and mutations

COL3A1 (also known as Collagen alpha-1(III) chain) is a human protein-coding gene encoding a collagen alpha-1(III) chain protein. Its type III collagen fibrils provide tensile support in arteries, bowel, uterus, skin, and other distensible connective tissues. Pathogenic variants cause vascular Ehlers-Danlos syndrome with marked arterial and hollow-organ fragility. This analysis covers 2,735 COL3A1 variants and mutations. Of these, 78% have computational variant effect predictions. Disease context includes Ehlers-Danlos syndrome, vascular type, polymicrogyria with or without vascular-type Ehlers-Danlos syndrome, and autosomal dominant Ehlers-Danlos syndrome, vascular type. Example COL3A1 variants include M1L, M2T, and M2V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable COL3A1 variants

Examples include M1L, M2T, M2V, M2L, M2R, M2K, S3R, S3S. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.